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ADDIN EN.CITE.DATA [10] که با افزايش خطر ابتلا به مقاومت انسولين و ديابت نوع 2 همراه است ADDIN EN.CITE <EndNote><Cite><Author>Cummings</Author><Year>2003</Year><RecNum>311</RecNum><DisplayText>[11]</DisplayText><record><rec-number>311</rec-number><foreign-keys><key app=”EN” db-id=”f55e2zwz4fax0newfdq5sw0hwwsvvw5dtwa5″>311</key></foreign-keys><ref-type name=”Journal Article”>17</ref-type><contributors><authors><author>Cummings, D. E.</author><author>Schwartz, M. W.</author></authors></contributors><auth-address>Department of Medicine, VA Puget Sound Health Care Sys– and Harborview Medical Center, University of Washington, Seattle, Washington 98195, USA. [email protected]</auth-address><titles><title>Genetics and pathophysiology of human obesity</title><secondary-title>Annual review of medicine</secondary-title><alt-title>Annu Rev Med</alt-title></titles><periodical><full-title>Annual review of medicine</full-title><abbr-1>Annu Rev Med</abbr-1></periodical><alt-periodical><full-title>Annual review of medicine</full-title><abbr-1>Annu Rev Med</abbr-1></alt-periodical><pages>453-71</pages><volume>54</volume><edition>2002/11/05</edition><keywords><keyword>Animals</keyword><keyword>Energy Metabolism/genetics/physiology</keyword><keyword>Genetic Predisposition to Disease/genetics</keyword><keyword>Ghrelin</keyword><keyword>Homeostasis/physiology</keyword><keyword>Humans</keyword><keyword>Hypothalamus/physiopathology</keyword><keyword>Leptin/physiology</keyword><keyword>Obesity/*genetics/physiopathology</keyword><keyword>Peptide Hormones/physiology</keyword><keyword>alpha-MSH/physiology</keyword></keywords><dates><year>2003</year></dates><isbn>0066-4219 (Print)&#xD;0066-4219 (Linking)</isbn><accession-num>12414915</accession-num><work-type>Review</work-type><urls><related-urls><url>http://www.ncbi.nlm.nih.gov/pubmed/12414915</url></related-urls></urls><electronic-resource-num>10.1146/annurev.med.54.101601.152403</electronic-resource-num><language>eng</language></record></Cite></EndNote>[11].
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